G7.iii · Cardiovascular Pharmacology

This viva will assess your knowledge of the cardiac action potential and anti-arrhythmic drugs. This image shows the phases of the cardiac muscle action potential. Describe the electrophysiological processes responsible for each phase. (Image removed from report).

This viva will explore your knowledge of anti-arrhythmic agents and the electrocardiogram. Briefly describe the phases of an action potential of the SA nodal / pacemaker cell. (Image removed from report.)

This viva will examine pacemaker cells and antiarrhythmic drugs. Explain the ionic currents responsible for spontaneous electrical activity of sinus node pacemaker cells.

This viva will explore your understanding of antibiotics and antiarrhythmics. Which classes of antibiotics are effective against Gram positive organisms?

This viva is on cardiac electrical physiology and anti-arrhythmic drugs. This is a normal trace obtained from the Lead II of the electrocardiogram. Describe the features of this trace. (Image removed from report.)

This viva tested knowledge of cardiovascular physiology. It discussed cardiac output, stroke volume, sarcomere length myocardial contractility, the role of calcium and then related pharmacology of drugs that increase cardiac contractility including catecholamines and digoxin.

This Viva tested knowledge of cardiovascular physiology, ECG and Antiarrhythmic pharmacology.

This Viva tested knowledge of on the physiology of calcium and the pharmacology of calcium channel antagonists. Although some candidates performed well, there was generally a lack of sufficient knowledge of the control of calcium.

This viva will examine knowledge of Calcium. Consider the normal levels of calcium in blood and factors which affect it. Subsequent discussion focused on calcium physiology and pharmacology of verapamil.

This Viva will test your knowledge of common β-blockers, and their pharmacology. How would you classify β Blockers? .../12 12. Subsequent questions explored knowledge of haemodynamic properties, receptor mediated effects, esmolol pharmacokinetics and sotalol as an antiarythmic drug. Following which, candidates were asked about the Pressure-Volume loop of the cardiac cycle and changes in that loop following administration of esmolol and labetalol.

This viva will assess your knowledge of the action potential of the ventricle, sinus node and the atrioventricular node. It will also assess knowledge of anti-arrhythmic drugs. From where does this action potential arise?